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[1]nflammation-induced microvascular insulin resistance is an early event in diet-induced obesity[J].Clin Sci (Lond)., 2015, 129 (12) : 1025-36.
[2]Globular adiponectin ameliorates metabolic insulin resistance via AMPK-mediated restoration of microvascular insulin responses.[J].J Physiol., 2014, 593 (17) : 4067-79.
[3]mTOR-mediated hyperphosphorylation of tau in the hippocampus is involved in cognitive deficits in streptozotocin-induced diabetic mice..Metab Brain Dis., 2014, 29 (3) : 729-36..
[4]Dysregulated mTOR-Dependent Signaling in Neurodegeneration or Carcinogenesis:Implication for Alzheimer’s Disease and Brain Tumors.J Alzheimers Dis.[J].J Alzheimers Dis., 2013, 37 (3) : 495-505.
[5]Tau pathology in diabetes mellitus[J]. Germany.Die Pharmazie, 2012: 649–652.
[6]Effect of genetic polymorphism of UCP2-866 G/A on repaglinide response in Chinese patients with type 2 diabetes[J]. Germany.Pharmazie., 2011: 74–79.
[7]NAMPT -3186C ⁄ T polymorphism affects repaglinide response in Chinese patients with Type 2 diabetes mellitus.Clinical and Experimental Pharmacology and Physiol, 2011, 38 (8) : 550-4.
[8]KCNJ11 Lys23Glu and TCF7L2 rs290487(C/T) polymorphisms affect therapeutic efficacy of repaglinide in Chinese patients with type 2 diabetes[J].Clin Pharmacol Ther., 2009, 87 (3) : 330-5..
[9]Liu ZQ., Zhou HH, Gong ZC, Yu M, Deng CS, Chen X, Wu J, Dai XP, Yin JY, Huang Q1.Association analysis of SLC30A8 rs13266634 and rs16889462 polymorphisms with type 2 diabetes mellitus and repaglinide response in Chinese patients.[J]. Europe.Eur J Clin Pharmacol., 2010, 66 (12) : 1207–1215.
[10]Plasma metabolic profiling of childhood obesity by GC/MS in conjunction with multivariate statistical analysis[J]. china.J Pharm Biomed Anal., 2010, 52 (2) : 265-72.
[11]Effects of UCP2 -866 G/A and ADRB3 Trp64Arg on rosiglitazone response in Chinese patients with Type 2 diabetes.[J]. british.Br J Clin Pharmacol., 2009, 68 (1) : 14-22.
[12]Rosiglitazone inhibits high glucose-induced apoptosis in human umbilical veinendothelial cells through PI3K/Akt/eNOS pathway[J]. canada.Can J Physiol Pharmacol., 2009, 87 (7) : 549-55.
[13]Liu ZQ., Zhou HH, Wang D, Zhang W, Yin JY, Hu PC, Gong ZC, Sun H, Wu J, Lin YG, Liu HL1.Impact of genetic polymorphisms of leptin and TNF-alpha on rosiglitazone response in Chinese patients with type 2 diabetes[J]. Europe.Eur J Clin Pharmacol., 2008, 64 (7) : 663-71..
[14]汶倩, 吴良燕, 王珊, 梁逸珍, 张婉, 吴静, 刘玲娇.多囊卵巢综合征患者超长链脂肪酸的变化及其与肥胖、胰岛素抵抗的关系[J]. 中国.中华糖尿病杂志, 2014, 6 (4) : 1-6.
[15]彭烈武, 雷闽湘, 莫娟, 吴静, 颜伟健.TNF-α 基因 G-308A 位点多态性 变化与儿童肥胖易感性的关系[J]. 中国.中华糖尿病杂志, 2012, 4 (1) : 23-27.
[16]吴静, 刘玲娇, 王珊.循证医学在内分泌学临床教学的应用[J]. 中国.基础医学教育, 2011, 13 (3) : 276-278.
[17]吴良燕, 吴 静.内分泌研究生教学中引入心理学课程的探索[J]. 中国.山西医科大学学报, 2010, 12 (9) : 926-928.
[18]周巧玲, 梁莉, 陈翔, 吴静.临床技能竞赛给诊断学教学带来的思考[J]. 中国.中国高等医学教育, 2014 (2) : 84-85.
[19]吴静 雷闽湘 孙志香 杨浩波.Searching of constructing an interdisciplinary innovative education system in endocrine postgraduate[J]. China.Researches inMedical Education, 2010, 9 (11) : 1448-1450.
[20]张国刚, 杨天伦, 梁莉, 陈翔, 周巧玲, 吴静.Diagnostics and internal medicine education reform exploration of eight - year program medical education[J]. China.Basic Medical Education, 2014, 16 (7) : 538-541.
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